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[1] According to the researchers TGF-beta1 induction of angiogenesis requires a rapid and transient apoptotic effect mediated by VEGF/VEGFR2. AHK-Cu, by enhancing levels of the Vascular Endothelial Growth Factor, may possibly activate fibroblasts and endothelial cells

Collectively, these results may highlight BPC 157s role as a modulator of apoptosis by suppressing key pro- apoptotic mediators, thereby shifting the balance toward cell survival

Am J Med Sci