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Cellular human GST monomers physically interact with various cellular proteins

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The results at 48 weeks were striking

This cellular energy crisis directly manifests as the profound physical fatigue, "brain fog," and post-exertional malaise (PEM) that patients experience

Although polymorphisms in genes such as NLRP3 and IL1B have not yet been comprehensively investigated, their central roles in mediating the inflammatory response to MSU crystals imply that genetic variations in these pathways may modulate the threshold at which asymptomatic hyperuricemia progresses to clinically significant tophus formation, by regulating the magnitude and duration of inflammatory responses